The N-terminal loop of IRAK-4 death domain regulates ordered assembly of the Myddosome signalling scaffold

نویسندگان

  • Anthony C. G. Dossang
  • Precious G. Motshwene
  • Yang Yang
  • Martyn F. Symmons
  • Clare E. Bryant
  • Satty Borman
  • Julie George
  • Alexander N. R. Weber
  • Nicholas J. Gay
چکیده

Activation of Toll-like receptors induces dimerization and the recruitment of the death domain (DD) adaptor protein MyD88 into an oligomeric post receptor complex termed the Myddosome. The Myddosome is a hub for inflammatory and oncogenic signaling and has a hierarchical arrangement with 6-8 MyD88 molecules assembling with exactly 4 of IRAK-4 and 4 of IRAK-2. Here we show that a conserved motif in IRAK-4 (Ser8-X-X-X-Arg12) is autophosphorylated and that the phosphorylated DD is unable to form Myddosomes. Furthermore a mutant DD with the phospho-mimetic residue Asp at this position is impaired in both signalling and Myddosome assembly. IRAK-4 Arg12 is also essential for Myddosome assembly and signalling and we propose that phosphorylated Ser8 induces the N-terminal loop to fold into an α-helix. This conformer is stabilised by an electrostatic interaction between phospho-Ser8 and Arg12 and would destabilise a critical interface between IRAK-4 and MyD88. Interestingly IRAK-2 does not conserve this motif and has an alternative interface in the Myddosome that requires Arg67, a residue conserved in paralogues, IRAK-1 and 3(M).

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2016